Whole-Genome Sequences of Chlamydia psittaci Strain HJ, Isolated from Meat Pigeons with Severe Respiratory Distress and High Mortality
نویسندگان
چکیده
The obligate intracellular Gram-negative bacterium Chlamydia psittaci causes systemic disease in psittacine birds, domestic poultry, and wild fowl. Importantly, C. psittaci may cause pneumonia, encephalitis, endocarditis, and even death in humans. The potential of pigeons as a source of human psittacosis is supported worldwide by relatively high seroconversion rates in the birds. This study reports the whole-genome sequencing of C. psittaci strain HJ, isolated from meat pigeons suffering from severe pneumonia and high mortality in 2013 in Hebei, China.
منابع مشابه
Free-ranging birds Captive birds Songbirds Free-ranging birds Captive birds Waterfowl Free-ranging birds Captive birds Herons and allies Free-ranging birds Captive birds Pigeons and doves Free-ranging birds Captive birds
Cause Chlamydiosis refers to an infection with organisms of the genus Chlamydia sp., which are bacteria that live within animal cells. Chlamydia psittaci is the species generally associated with this disease in birds. The severity of the disease differs by the strain of C. psittaci and the susceptibility of different species of birds. As a result, chlamydiosis may range from an inapparent infec...
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Chlamydia psittaci is a highly prevalent avian pathogen and the cause of a potentially lethal zoonosis, causing life-threatening pneumonia in humans. We report the genome sequences of C. psittaci 6BC, the prototype strain of the species, and C. psittaci Cal10, a widely used laboratory strain.
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BACKGROUND AND OBJECTIVE Chlamydophila psittaci is a lethal bacterium that causes endemic avian chlamydiosis, and respiratory psittacosis. Laboratory diagnosis of Chlamydophila psittaci is difficult by culture. This study was design to investigate the presence of Chlamydophila psittaci in collected pharyngeal swabs from asyptomatic pigeons by PCR. MATERIALS AND METHODS Pharyngeal samples from...
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عنوان ژورنال:
دوره 3 شماره
صفحات -
تاریخ انتشار 2015